That's why we can't stop eating.
, they developed a line of mice that expresses CRTC1 normally aside from in MC4R-expressing neurons where it is blocked.
The animals lacking CRTC1 in MC4R-expressing neurons did not differ from control mice in terms of body weight when given a regular diet. The CRTC1-deficient animals who overate were noticeably more obese than the control mice and eventually acquired diabetes when they were fed a high-fat diet.“This study has revealed the role that the CRTC1 gene plays in the brain, and part of the mechanism that stops us from overeating high-calorie, fatty, and sugary foods,” said Professor Matsumura.
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