Take that, cancer!
In a nutshell, the treatment works by drawing a protein of the cancer causing KRAS gene, which is believed to be responsible for a full quarter of all tumors, to the cancer cell’s surface.
The problem is that it’s very hard for drugs to bind to a mutated, cancer-causing KRAS protein’s surface — and that’s assuming you can even reach the protein, because most of them reside inside the cell, where it avoids detection from the body’s immune system. "This mutated protein is usually flying under the radar because it’s so similar to the healthy protein," said Charles Craik, one of the study's authors and a professor of pharmaceutical chemistry at UCSF, in the same release. "But when you attach this drug to it, it gets spotted right away."
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